2005). These effects weren't shocking if we look at that PEA is really a improperly water?soluble material, that may limit its oral absorption and bioavailability, but they could also suggest a short?lived action of PEA, in arrangement with The point that this compound is degraded by two distinct hydrolases, that is definitely, NAAA and FAAH. After
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2015). These reports, taken alongside one another, counsel that, although a immediate activation of GPR55 or PPAR?? occurs, PEA can produce its anti?inflammatory motion while in the intestine also by using indirect activation of CB1 and CB2 receptors, probably as a consequence of the ability of this compound to potentiate the motion of endocannabin
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